Year 2007,
Volume: 12 Issue: 1, 79 - 80, 01.02.2007
Abstract
The patient, who was diagnosed epilepsy eight years ago, has been observed since then. The patient has been using carbamezapine for the last three years. He who did not have any seizures in the last year was clinically diagnosed status epilepticus. Status epilepticus was determined to have resulted from severe hypocalcemia. ©2007, Firat University, Medical Faculty
Keywords
References
- AndersenRS, ValeurN. Hyperparathyroidism secondary to anticonvulsant treatment. UgeskrLaeger 2001; 163: 6288-6289.
- Zdrojewicz Z, Dubinski A, Jonek A, Tomanek N, Krajewski W Tetanic crisis and antiepileptic drugs. A case report. Neuro Endocrinol Lett 2005; 26: 317-319.
- Pack AM Bone disease in epilepsy. Curr Neurol Neurosci Rep 2004; 4: 329-334.
- Ali FE, Al-Bustan MA, Al-Busairi WA, Al-Mulla FA. Loss of seizure control due to anticonvulsant-induced hypocalcemia. Ann Pharmacother 2004; 38: 1002-1005.
- Kayemba Kay'S S, Beust M, Aboulghit H, Voisin M, Mourtada A. Carbamazepine and vigabatrin in epileptic pregnant woman and side effects in the newborn infant. Arch Pediatr 1997; 4: 975- 978.
- Hoikka V, Alhava EM, Karjalainen P, et al. Carbamazepine and bone mineral metabolism. Acta Neurol Scand 1984 ; 70: 77-80.
- Boran P, Yılmaz Y, Turan S, Bereket A. Karbameazepin ve Valproat monoterapilerinin tiroid fonksiyonu, lipid ve kemik metabolizması üzerine etkileri. Çocuk Dergisi (Logos) 2004; 4: 157-162.
- Aka S, Erverdi D, Göğcü Ş, Demirkol A, Yavrucu S, Özgüner A. Antiepileptik ilaç kullanan çocuklarda serum kalsiyum düzeyleri. PTT Hastanesi Tıp Dergisi 2000; 22: 154-156.
- LA Fıtzpatrick. Pathophysiology of bone loss in patients receiving anticonvulsant therapy. Epilepsi & Behavior 2004; 5: 3- 15.
- Kabul Tarihi: 15.12.2006
Year 2007,
Volume: 12 Issue: 1, 79 - 80, 01.02.2007
Abstract
Kliniğimizde sekiz yıldır epilepsi tanısı ile izlenen hasta, son üç yıldır karbamazepin kullanıyordu ve son bir yıldır da hiç nöbeti yokken status epileptikus kliniği ile başvurdu. Status epileptikusun ağır hipokalsemiden kaynaklandığı belirlendi. ©2007, Fırat Üniversitesi, Tıp Fakültesi
Keywords
References
- AndersenRS, ValeurN. Hyperparathyroidism secondary to anticonvulsant treatment. UgeskrLaeger 2001; 163: 6288-6289.
- Zdrojewicz Z, Dubinski A, Jonek A, Tomanek N, Krajewski W Tetanic crisis and antiepileptic drugs. A case report. Neuro Endocrinol Lett 2005; 26: 317-319.
- Pack AM Bone disease in epilepsy. Curr Neurol Neurosci Rep 2004; 4: 329-334.
- Ali FE, Al-Bustan MA, Al-Busairi WA, Al-Mulla FA. Loss of seizure control due to anticonvulsant-induced hypocalcemia. Ann Pharmacother 2004; 38: 1002-1005.
- Kayemba Kay'S S, Beust M, Aboulghit H, Voisin M, Mourtada A. Carbamazepine and vigabatrin in epileptic pregnant woman and side effects in the newborn infant. Arch Pediatr 1997; 4: 975- 978.
- Hoikka V, Alhava EM, Karjalainen P, et al. Carbamazepine and bone mineral metabolism. Acta Neurol Scand 1984 ; 70: 77-80.
- Boran P, Yılmaz Y, Turan S, Bereket A. Karbameazepin ve Valproat monoterapilerinin tiroid fonksiyonu, lipid ve kemik metabolizması üzerine etkileri. Çocuk Dergisi (Logos) 2004; 4: 157-162.
- Aka S, Erverdi D, Göğcü Ş, Demirkol A, Yavrucu S, Özgüner A. Antiepileptik ilaç kullanan çocuklarda serum kalsiyum düzeyleri. PTT Hastanesi Tıp Dergisi 2000; 22: 154-156.
- LA Fıtzpatrick. Pathophysiology of bone loss in patients receiving anticonvulsant therapy. Epilepsi & Behavior 2004; 5: 3- 15.
- Kabul Tarihi: 15.12.2006
There are 10 citations in total.
Details
| Primary Language | Turkish |
|---|---|
| Journal Section | Articles |
| Authors | |
| Publication Date | February 1, 2007 |
| Published in Issue | Year 2007 Volume: 12 Issue: 1 |