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Anatomical Examination of Pituitary Adenoma Compressing the Oculomotor Nerve

Year 2015, Volume: 5 Issue: 1-Ek, 100 - 77, 26.08.2015
https://doi.org/10.16899/ctd.91054

Abstract

Oculomotor nerve palsy may occur as a result of benign or malignant pathologies such as trauma, aneurysm and tumor. Ptosis, mydriasis and diplopia may be seen depending on the affected nerve fibers and nucleus. While visual field defects such as bitemporal hemianopsia usually were seen in the initial stages of the pituitary adenoma which may be important causes of the oculomotor nerve palsy, generally third cranial nerve palsy symptoms were seen in advanced stages. In this case report, the oculomotor nerve palsy which caused by pituitary adenomas were presented.

 

 

References

  • Gottlieb M, Kogan A, Kimball D. Intracranial tuberculoma presenting as an isolated oculomotor nerve paresis. J Emerg Med. 2015;48:e1-4.
  • Komurcu HF, Ayberk G, Ozveren MF, Anlar O. Pituitary adenoma apoplexy presenting with bilateral third nerve palsy and bilateral proptosis: a case report. Med Princ Pract. 2012;21:285-7.
  • Kobayashi H, Kawabori M, Terasaka S, Murata J, Houkin K. A possible mechanism of isolated oculomotor nerve palsy by apoplexy of pituitary adenoma without cavernous sinus invasion: a report of two cases. Acta Neurochir. 2011;153:2453-6.
  • Fonksiyonel Nöroanatomi. Doğan Taner. ODTÜ Yayıncılık, 6. Basım 2007, Ankara (Sayfa 141-143, 238)
  • Chugh JP, J Prachi, Chouhan RS, Rathi A. Third Nerve Palsy: An Overview. Indian Journal of Clinical Practice. 2012;22:12.
  • Clauser L, Tieghi R, Galiè M. Palpebralptosis: clinical classification, differential diagnosis, and surgical guidelines: an overview. J Craniofac Surg. 2006;17:246-54.
  • Gray’s Anatomy, Editor in Chief: Susan Standring 2008 Churchill Livingstone Elsevier Fortieth Edition (p 290, 291, 659, 661)
  • Bhatti MT, Eisenschenk S, Roper SN, Guy JR. Superior divisional third cranial nerve paresis: clinical and anatomical observations of 2 unique cases. Arch Neurol. 2006 May;63(5):771-6.
  • Terasaka S, Murata J, Houkin K. A possible mechanism of isolated oculomotor nerve palsy by apoplexy of pituitary adenoma without cavernous sinus invasion: a report of two cases. Acta Neurochir. 2011;153:2453-6.

Okulomotor Sinire Bası Yapan Hipofiz Adenomunun Anatomik Olarak İncelenmesi

Year 2015, Volume: 5 Issue: 1-Ek, 100 - 77, 26.08.2015
https://doi.org/10.16899/ctd.91054

Abstract

Okulomotor sinir felci travma, anevrizma, tümörler gibi benign veya malign olabilen patolojiler sonucunda gelişebilir.  Etkilenen çekirdeğe ve sinir liflerine bağlı olarak  pitozis, midriazis, diplopi görülebilir. Okulomotor sinir felcinin önemli nedenlerinden biri olabilen hipofiz adenomlarında genellikle başlangıçta bitemporal hemianopsi gibi görme defektleri görülürken genellikle ileri safhalarda üçüncü kranial sinir felci bulguları görülebilir. Bu olgu sunumunda, hipofiz adenomunun sebep olduğu okulomotor sinir felci sunuldu.

 

References

  • Gottlieb M, Kogan A, Kimball D. Intracranial tuberculoma presenting as an isolated oculomotor nerve paresis. J Emerg Med. 2015;48:e1-4.
  • Komurcu HF, Ayberk G, Ozveren MF, Anlar O. Pituitary adenoma apoplexy presenting with bilateral third nerve palsy and bilateral proptosis: a case report. Med Princ Pract. 2012;21:285-7.
  • Kobayashi H, Kawabori M, Terasaka S, Murata J, Houkin K. A possible mechanism of isolated oculomotor nerve palsy by apoplexy of pituitary adenoma without cavernous sinus invasion: a report of two cases. Acta Neurochir. 2011;153:2453-6.
  • Fonksiyonel Nöroanatomi. Doğan Taner. ODTÜ Yayıncılık, 6. Basım 2007, Ankara (Sayfa 141-143, 238)
  • Chugh JP, J Prachi, Chouhan RS, Rathi A. Third Nerve Palsy: An Overview. Indian Journal of Clinical Practice. 2012;22:12.
  • Clauser L, Tieghi R, Galiè M. Palpebralptosis: clinical classification, differential diagnosis, and surgical guidelines: an overview. J Craniofac Surg. 2006;17:246-54.
  • Gray’s Anatomy, Editor in Chief: Susan Standring 2008 Churchill Livingstone Elsevier Fortieth Edition (p 290, 291, 659, 661)
  • Bhatti MT, Eisenschenk S, Roper SN, Guy JR. Superior divisional third cranial nerve paresis: clinical and anatomical observations of 2 unique cases. Arch Neurol. 2006 May;63(5):771-6.
  • Terasaka S, Murata J, Houkin K. A possible mechanism of isolated oculomotor nerve palsy by apoplexy of pituitary adenoma without cavernous sinus invasion: a report of two cases. Acta Neurochir. 2011;153:2453-6.
There are 9 citations in total.

Details

Primary Language English
Journal Section Case Report
Authors

Murat Uysal

Publication Date August 26, 2015
Published in Issue Year 2015 Volume: 5 Issue: 1-Ek

Cite

APA Uysal, M. (2015). Anatomical Examination of Pituitary Adenoma Compressing the Oculomotor Nerve. Çağdaş Tıp Dergisi, 5(1-Ek), 100-77. https://doi.org/10.16899/ctd.91054
AMA Uysal M. Anatomical Examination of Pituitary Adenoma Compressing the Oculomotor Nerve. J Contemp Med. August 2015;5(1-Ek):100-77. doi:10.16899/ctd.91054
Chicago Uysal, Murat. “Anatomical Examination of Pituitary Adenoma Compressing the Oculomotor Nerve”. Çağdaş Tıp Dergisi 5, no. 1-Ek (August 2015): 100-77. https://doi.org/10.16899/ctd.91054.
EndNote Uysal M (August 1, 2015) Anatomical Examination of Pituitary Adenoma Compressing the Oculomotor Nerve. Çağdaş Tıp Dergisi 5 1-Ek 100–77.
IEEE M. Uysal, “Anatomical Examination of Pituitary Adenoma Compressing the Oculomotor Nerve”, J Contemp Med, vol. 5, no. 1-Ek, pp. 100–77, 2015, doi: 10.16899/ctd.91054.
ISNAD Uysal, Murat. “Anatomical Examination of Pituitary Adenoma Compressing the Oculomotor Nerve”. Çağdaş Tıp Dergisi 5/1-Ek (August 2015), 100-77. https://doi.org/10.16899/ctd.91054.
JAMA Uysal M. Anatomical Examination of Pituitary Adenoma Compressing the Oculomotor Nerve. J Contemp Med. 2015;5:100–77.
MLA Uysal, Murat. “Anatomical Examination of Pituitary Adenoma Compressing the Oculomotor Nerve”. Çağdaş Tıp Dergisi, vol. 5, no. 1-Ek, 2015, pp. 100-77, doi:10.16899/ctd.91054.
Vancouver Uysal M. Anatomical Examination of Pituitary Adenoma Compressing the Oculomotor Nerve. J Contemp Med. 2015;5(1-Ek):100-77.