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Sık Ventriküler Erken Vuruların Sebep Olduğu Dekompanse Kalp Yetmezliği

Year 2015, Volume: 68 Issue: 1, 37 - 40, 10.08.2015

Abstract

Asymptomatic premature ventricular beats are most common presentation form of arrhythmia
observed in patients with or without structural heart disease. In long term, frequent premature
ventricular beats can lead left ventricule failure in structurally normal heart. Here, we
reported a case of premature ventricular complex-induced cardiomyopathy who applied to
emergency department with acute heart failure and recovered promptly after catheter ablation
of related arrhytmic focus.

Ethical Statement

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Supporting Institution

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Project Number

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Thanks

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References

  • 1- Katritsis DG, Siontis GC, Camm AJ. Prognostic significance of ambulatory ECG monitoring for ventricular arrhythmias. Prog Cardiovasc Dis. 2013;56:133-142.
  • 2- Simpson RJ Jr, Cascio WE, Schreiner PJ, et al. Prevalence of premature ventricular contractions in a population of African American and white men and women: The Atherosclerosis Risk In Communities (ARIC) study. Am Heart J 2002;143:535–540.
  • 3- Ephrem G, Levine M, Friedmann P, Schweitzer P. The prognostic significance frequency and morphology of premature ventricular complexes during ambulatory holter monitoring. Ann Noninvasive Electrocardiol. 2013;18:118-125
  • 4- Bikkina M, Larson MG, Levy D. Prognostic implications of asymptomatic ventricular arrhythmias: The Framingham Heart Study. Ann Intern Med 1992;117:990– 996.
  • 5- Sheldon SH, Gard JJ, Asirvatham SJ. Premature Ventricular Contractions and Non-sustained Ventricular Tachycardia: Association with Sudden Cardiac Death, Risk Stratification, and Management Strategies. Indian Pacing Electrophysiol J. 2010;10:357-371.
  • 6- Duffee DF, Shen WK, Smith HC. Suppression of frequent premature ventricular contractions and improvement of left ventricular function in patients with presumed idiopathic dilated cardiomyopathy. Mayo Clin Proc 1998;73:430–433.
  • 7- Lee GK, Klarich KW, Grogan M, Cha YM. Premature ventricular contractioninduced cardiomyopathy: a treatable condition. Circ Arrhythm Electrophysiol. 2012;5:229-236
  • 8- Winkens RA, Höppener PF, Kragten JA, et al.Are premature ventricular contractions always harmless? Eur J Gen Pract. 2013 Nov 28. [Epub ahead of print]
  • 9- Yokokawa M, Kim HM, Good E, et al. Relation of symptoms and symptom duration to premature ventricular complexinduced cardiomyopathy. Heart Rhythm. 2012;9:92- 95.
  • 10- Hasdemir C, Kartal Y, Simsek E ve ark. Time course of recovery of left ventricular systolic dysfunction in patients with premature ventricular contractioninduced cardiomyopathy. Pacing Clin Electrophysiol. 2013;36:612-617.
  • 11- Yokokawa M, Good E, Crawford T, Chugh A, Pelosi F Jr, Latchamsetty R, Jongnarangsin K, Armstrong W, Ghanbari H, Oral H, Morady F, Bogun F. Recovery from left ventricular dysfunction after ablation of frequent premature ventricular complexes. Heart Rhythm. 2013;10:172-175.
  • 12- Del Carpio Munoz F, Syed FF, Noheria A, et al. Characteristics of premature ventricular complexes as correlates of reduced left ventricular systolic function: study of the burden, duration, coupling interval, morphology and site of origin of PVCs. J Cardiovasc Electrophysiol. 2011;22:791-798.
  • 13- Chugh SS, Shen WK, Luria DM, et al. First evidence of premature ventricular complex-induced cardiomyopathy: A potentially reversible cause of heart failure. J Cardiovasc Electrophysiol 2000;11: 328 – 329
  • 14- Mann SA, Castro ML, Ohanian M, Guo G, Zodgekar P, Sheu A et al. R222Q SCN5A mutation is associated with reversible ventricular ectopy and dilated cardiomyopathy. J Am Coll Cardiol. 2012;60:1566-1573.

Frequent Ventricular Premature Complex-Induced Decompensated Heart Failure

Year 2015, Volume: 68 Issue: 1, 37 - 40, 10.08.2015

Abstract

Project Number

-

References

  • 1- Katritsis DG, Siontis GC, Camm AJ. Prognostic significance of ambulatory ECG monitoring for ventricular arrhythmias. Prog Cardiovasc Dis. 2013;56:133-142.
  • 2- Simpson RJ Jr, Cascio WE, Schreiner PJ, et al. Prevalence of premature ventricular contractions in a population of African American and white men and women: The Atherosclerosis Risk In Communities (ARIC) study. Am Heart J 2002;143:535–540.
  • 3- Ephrem G, Levine M, Friedmann P, Schweitzer P. The prognostic significance frequency and morphology of premature ventricular complexes during ambulatory holter monitoring. Ann Noninvasive Electrocardiol. 2013;18:118-125
  • 4- Bikkina M, Larson MG, Levy D. Prognostic implications of asymptomatic ventricular arrhythmias: The Framingham Heart Study. Ann Intern Med 1992;117:990– 996.
  • 5- Sheldon SH, Gard JJ, Asirvatham SJ. Premature Ventricular Contractions and Non-sustained Ventricular Tachycardia: Association with Sudden Cardiac Death, Risk Stratification, and Management Strategies. Indian Pacing Electrophysiol J. 2010;10:357-371.
  • 6- Duffee DF, Shen WK, Smith HC. Suppression of frequent premature ventricular contractions and improvement of left ventricular function in patients with presumed idiopathic dilated cardiomyopathy. Mayo Clin Proc 1998;73:430–433.
  • 7- Lee GK, Klarich KW, Grogan M, Cha YM. Premature ventricular contractioninduced cardiomyopathy: a treatable condition. Circ Arrhythm Electrophysiol. 2012;5:229-236
  • 8- Winkens RA, Höppener PF, Kragten JA, et al.Are premature ventricular contractions always harmless? Eur J Gen Pract. 2013 Nov 28. [Epub ahead of print]
  • 9- Yokokawa M, Kim HM, Good E, et al. Relation of symptoms and symptom duration to premature ventricular complexinduced cardiomyopathy. Heart Rhythm. 2012;9:92- 95.
  • 10- Hasdemir C, Kartal Y, Simsek E ve ark. Time course of recovery of left ventricular systolic dysfunction in patients with premature ventricular contractioninduced cardiomyopathy. Pacing Clin Electrophysiol. 2013;36:612-617.
  • 11- Yokokawa M, Good E, Crawford T, Chugh A, Pelosi F Jr, Latchamsetty R, Jongnarangsin K, Armstrong W, Ghanbari H, Oral H, Morady F, Bogun F. Recovery from left ventricular dysfunction after ablation of frequent premature ventricular complexes. Heart Rhythm. 2013;10:172-175.
  • 12- Del Carpio Munoz F, Syed FF, Noheria A, et al. Characteristics of premature ventricular complexes as correlates of reduced left ventricular systolic function: study of the burden, duration, coupling interval, morphology and site of origin of PVCs. J Cardiovasc Electrophysiol. 2011;22:791-798.
  • 13- Chugh SS, Shen WK, Luria DM, et al. First evidence of premature ventricular complex-induced cardiomyopathy: A potentially reversible cause of heart failure. J Cardiovasc Electrophysiol 2000;11: 328 – 329
  • 14- Mann SA, Castro ML, Ohanian M, Guo G, Zodgekar P, Sheu A et al. R222Q SCN5A mutation is associated with reversible ventricular ectopy and dilated cardiomyopathy. J Am Coll Cardiol. 2012;60:1566-1573.
There are 14 citations in total.

Details

Primary Language English
Subjects Cardiology
Journal Section Case Report
Authors

Hüseyin Ede 0000-0003-1218-257X

Veysel Kutay Vurgun 0000-0001-8624-2809

Ali Timuçin Altın This is me 0000-0001-8562-9673

Başar Candemir 0000-0003-2657-7601

Ömer Akyürek 0000-0002-8100-1800

Çetin Erol 0000-0001-7396-3818

Project Number -
Publication Date August 10, 2015
Published in Issue Year 2015 Volume: 68 Issue: 1

Cite

APA Ede, H., Vurgun, V. K., Altın, A. T., … Candemir, B. (2015). Sık Ventriküler Erken Vuruların Sebep Olduğu Dekompanse Kalp Yetmezliği. Ankara Üniversitesi Tıp Fakültesi Mecmuası, 68(1), 37-40.
AMA Ede H, Vurgun VK, Altın AT, Candemir B, Akyürek Ö, Erol Ç. Sık Ventriküler Erken Vuruların Sebep Olduğu Dekompanse Kalp Yetmezliği. Ankara Üniversitesi Tıp Fakültesi Mecmuası. August 2015;68(1):37-40.
Chicago Ede, Hüseyin, Veysel Kutay Vurgun, Ali Timuçin Altın, Başar Candemir, Ömer Akyürek, and Çetin Erol. “Sık Ventriküler Erken Vuruların Sebep Olduğu Dekompanse Kalp Yetmezliği”. Ankara Üniversitesi Tıp Fakültesi Mecmuası 68, no. 1 (August 2015): 37-40.
EndNote Ede H, Vurgun VK, Altın AT, Candemir B, Akyürek Ö, Erol Ç (August 1, 2015) Sık Ventriküler Erken Vuruların Sebep Olduğu Dekompanse Kalp Yetmezliği. Ankara Üniversitesi Tıp Fakültesi Mecmuası 68 1 37–40.
IEEE H. Ede, V. K. Vurgun, A. T. Altın, B. Candemir, Ö. Akyürek, and Ç. Erol, “Sık Ventriküler Erken Vuruların Sebep Olduğu Dekompanse Kalp Yetmezliği”, Ankara Üniversitesi Tıp Fakültesi Mecmuası, vol. 68, no. 1, pp. 37–40, 2015.
ISNAD Ede, Hüseyin et al. “Sık Ventriküler Erken Vuruların Sebep Olduğu Dekompanse Kalp Yetmezliği”. Ankara Üniversitesi Tıp Fakültesi Mecmuası 68/1 (August2015), 37-40.
JAMA Ede H, Vurgun VK, Altın AT, Candemir B, Akyürek Ö, Erol Ç. Sık Ventriküler Erken Vuruların Sebep Olduğu Dekompanse Kalp Yetmezliği. Ankara Üniversitesi Tıp Fakültesi Mecmuası. 2015;68:37–40.
MLA Ede, Hüseyin et al. “Sık Ventriküler Erken Vuruların Sebep Olduğu Dekompanse Kalp Yetmezliği”. Ankara Üniversitesi Tıp Fakültesi Mecmuası, vol. 68, no. 1, 2015, pp. 37-40.
Vancouver Ede H, Vurgun VK, Altın AT, Candemir B, Akyürek Ö, Erol Ç. Sık Ventriküler Erken Vuruların Sebep Olduğu Dekompanse Kalp Yetmezliği. Ankara Üniversitesi Tıp Fakültesi Mecmuası. 2015;68(1):37-40.