Tefferi A. Classification, diagnosis and management of myeloproliferative disorders in the JAK2V617F era. Hematology Am Soc Hematol Educ Program 2006;240–5.
James C, Ugo V, Le Couedic JP et al. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera. Nature 2005;434:1144–8.
Wernig G, Mercher T, Okabe R, Levine RL, Lee BH, Gilliland DG. Expression of Jak2V617F causes a polycthemia vera-like disease with associated myelofibrosis in a murine bone marrow transplant model. Blood 2006;107:4274–81.
Jelinek J, Oki Y, Gharibyan V et al. JAK2 mutation 1849G>T is rare in acute leukemias but can be found in CMML, Philadelphia chromosome-negative CML, and megakaryocytic leukemia. Blood 2005;106:3370–3.
Bock O, Büsche G, Koop C, Schröter S, Buhr T, Kreipe H. Detection of the single hotspot mutation in the JH2 pseudokinase domain of janus kinase 2 in bone marrow trephine biopsies derived from chronic myeloproliferative disorders. Journal of Molecular Diagnostics 2006; 8:170-7.
Krämer A. JAK2-V617F and BCR-ABL-double Jeopardy? Leukemia Res 2008;32:1489–90.
Pardini S, Fozza C, Contini S et al. A case of coexistence between JAK2V617F and BCR/ABL. Eur J Hematol 2008; 81.75–6.
Inami M, Inokuchi K, Okabe M et al. Polycthemia associated with the JAK2V617F mutation emerged during treatment of chronic mylogeneous leukemia. Leukemia 2007;21:1103–4.
Cambier N, Renneville A, Cazaentre T et al. JAK2V617Fpositive polycythemia vera and philadelphia chromosome-positive chronic myeloid leukemia: one patient with two distinct myeloproliferative disorders. Leukemia 2008;22:1454–5.
Hussein K, Bock O, Seegers E et al. Myelofibrosis evolving during imatinib treatment of a chronic myeloproliferative disorder with coexisting BCR-ABL translocation and JAK2V617F mutation. Blood 2007;109:4106–7.
Krämer A, Reiter A, Kruth J et al. JAK2-V617F mutation in a patient with Philadelphia-chromosome-positive chronic myeloid leukemia. Lancet 2007;8:658–60.
Veronese L, Tchirkov A, Richard-Pebrel C et al. A thrombocytosis occuring in Philadelphia positive CML in molecular response to imatinib can reveal an underlying JAK2V617F myeloproliferative neoplasm. Leuk Res 2010;34:94–6.
Campiotti L, Appio L, Solbiati F, AgenoW, Venco A. JAK2V617F mutation and Philadelphia positive chronic myeloid leukemia. Leukemia Research 2009; 33.212–3.
Jallades L, Hateyye S, Tigaud I et al. Emergence of therapy-unrelated CML on a backgraund of BCR-ABL-negative JAK2V617F-positive chronic idiopathic myelofibrosis. Leuk Res 2008;32:1608–10.
Bornhäuser M, Mohr B, Oelschlaegel U et al. Concurrent JAK2(V617F) mutation and BCR-ABL translocation within committed myeloid progenitors in myelofibrosis. Leukemia 2007; 21:1824–6.
Curtin NJ, Campbell PJ, Green AR. The Philadelphia translocation and pre-existing myeloproliferative disorders. Br J Haematol 2005;128:730–6.
Coexistance of JAK2V617F mutation and BCR/ABL translocation in one patient
Year 2010,
Volume: 1 Issue: 2, 138 - 140, 01.06.2010
liland DG. Expression of Jak2V617F causes a polycthemia vera-like disease with associated myelofibrosis in a murine bone marrow transplant model. Blood 2006;107:4274–81. 4. Jelinek J, Oki Y, Gharibyan V et al. JAK2 mutation 1849G>T is rare in acute leukemias but can be found in CMML, Phil- adelphia chromosome-negative CML, and megakaryocytic leukemia. Blood 2005;106:3370–3
Tefferi A. Classification, diagnosis and management of myeloproliferative disorders in the JAK2V617F era. Hematology Am Soc Hematol Educ Program 2006;240–5.
James C, Ugo V, Le Couedic JP et al. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera. Nature 2005;434:1144–8.
Wernig G, Mercher T, Okabe R, Levine RL, Lee BH, Gilliland DG. Expression of Jak2V617F causes a polycthemia vera-like disease with associated myelofibrosis in a murine bone marrow transplant model. Blood 2006;107:4274–81.
Jelinek J, Oki Y, Gharibyan V et al. JAK2 mutation 1849G>T is rare in acute leukemias but can be found in CMML, Philadelphia chromosome-negative CML, and megakaryocytic leukemia. Blood 2005;106:3370–3.
Bock O, Büsche G, Koop C, Schröter S, Buhr T, Kreipe H. Detection of the single hotspot mutation in the JH2 pseudokinase domain of janus kinase 2 in bone marrow trephine biopsies derived from chronic myeloproliferative disorders. Journal of Molecular Diagnostics 2006; 8:170-7.
Krämer A. JAK2-V617F and BCR-ABL-double Jeopardy? Leukemia Res 2008;32:1489–90.
Pardini S, Fozza C, Contini S et al. A case of coexistence between JAK2V617F and BCR/ABL. Eur J Hematol 2008; 81.75–6.
Inami M, Inokuchi K, Okabe M et al. Polycthemia associated with the JAK2V617F mutation emerged during treatment of chronic mylogeneous leukemia. Leukemia 2007;21:1103–4.
Cambier N, Renneville A, Cazaentre T et al. JAK2V617Fpositive polycythemia vera and philadelphia chromosome-positive chronic myeloid leukemia: one patient with two distinct myeloproliferative disorders. Leukemia 2008;22:1454–5.
Hussein K, Bock O, Seegers E et al. Myelofibrosis evolving during imatinib treatment of a chronic myeloproliferative disorder with coexisting BCR-ABL translocation and JAK2V617F mutation. Blood 2007;109:4106–7.
Krämer A, Reiter A, Kruth J et al. JAK2-V617F mutation in a patient with Philadelphia-chromosome-positive chronic myeloid leukemia. Lancet 2007;8:658–60.
Veronese L, Tchirkov A, Richard-Pebrel C et al. A thrombocytosis occuring in Philadelphia positive CML in molecular response to imatinib can reveal an underlying JAK2V617F myeloproliferative neoplasm. Leuk Res 2010;34:94–6.
Campiotti L, Appio L, Solbiati F, AgenoW, Venco A. JAK2V617F mutation and Philadelphia positive chronic myeloid leukemia. Leukemia Research 2009; 33.212–3.
Jallades L, Hateyye S, Tigaud I et al. Emergence of therapy-unrelated CML on a backgraund of BCR-ABL-negative JAK2V617F-positive chronic idiopathic myelofibrosis. Leuk Res 2008;32:1608–10.
Bornhäuser M, Mohr B, Oelschlaegel U et al. Concurrent JAK2(V617F) mutation and BCR-ABL translocation within committed myeloid progenitors in myelofibrosis. Leukemia 2007; 21:1824–6.
Curtin NJ, Campbell PJ, Green AR. The Philadelphia translocation and pre-existing myeloproliferative disorders. Br J Haematol 2005;128:730–6.
Yokuş, O., Kurnaz, F., Balçık, Ö. Ş., Uz, B., et al. (2010). Coexistance of JAK2V617F mutation and BCR/ABL translocation in one patient. Journal of Clinical and Experimental Investigations, 1(2), 138-140. https://doi.org/10.5799/ahinjs.01.2010.02.0029
AMA
Yokuş O, Kurnaz F, Balçık ÖŞ, Uz B, Albayrak M. Coexistance of JAK2V617F mutation and BCR/ABL translocation in one patient. J Clin Exp Invest. June 2010;1(2):138-140. doi:10.5799/ahinjs.01.2010.02.0029
Chicago
Yokuş, Osman, Fatih Kurnaz, Özlem Şahin Balçık, Burak Uz, and Murat Albayrak. “Coexistance of JAK2V617F Mutation and BCR/ABL Translocation in One Patient”. Journal of Clinical and Experimental Investigations 1, no. 2 (June 2010): 138-40. https://doi.org/10.5799/ahinjs.01.2010.02.0029.
EndNote
Yokuş O, Kurnaz F, Balçık ÖŞ, Uz B, Albayrak M (June 1, 2010) Coexistance of JAK2V617F mutation and BCR/ABL translocation in one patient. Journal of Clinical and Experimental Investigations 1 2 138–140.
IEEE
O. Yokuş, F. Kurnaz, Ö. Ş. Balçık, B. Uz, and M. Albayrak, “Coexistance of JAK2V617F mutation and BCR/ABL translocation in one patient”, J Clin Exp Invest, vol. 1, no. 2, pp. 138–140, 2010, doi: 10.5799/ahinjs.01.2010.02.0029.
ISNAD
Yokuş, Osman et al. “Coexistance of JAK2V617F Mutation and BCR/ABL Translocation in One Patient”. Journal of Clinical and Experimental Investigations 1/2 (June 2010), 138-140. https://doi.org/10.5799/ahinjs.01.2010.02.0029.
JAMA
Yokuş O, Kurnaz F, Balçık ÖŞ, Uz B, Albayrak M. Coexistance of JAK2V617F mutation and BCR/ABL translocation in one patient. J Clin Exp Invest. 2010;1:138–140.
MLA
Yokuş, Osman et al. “Coexistance of JAK2V617F Mutation and BCR/ABL Translocation in One Patient”. Journal of Clinical and Experimental Investigations, vol. 1, no. 2, 2010, pp. 138-40, doi:10.5799/ahinjs.01.2010.02.0029.
Vancouver
Yokuş O, Kurnaz F, Balçık ÖŞ, Uz B, Albayrak M. Coexistance of JAK2V617F mutation and BCR/ABL translocation in one patient. J Clin Exp Invest. 2010;1(2):138-40.