BibTex RIS Kaynak Göster

Chorea due to basal ganglia involvement in a uremic diabetic patient

Yıl 2014, Cilt: 39 Sayı: 2, 353 - 356, 22.07.2014
https://doi.org/10.17826/cutf.38301

Öz

Syndromes associated with acute bilateral lesions of the basal ganglia in diabetic uremic patients are uncommon. Uremic encephalopathy is typical of patients showing cortical involvement, with symptoms including confusion, seizures, tremors, or myoclonus. Whenever basal ganglia are anatomically involved, movement disorders arise, including chorea. In this article we present a case with basal ganglia involvement in a uremic diabetic patient causes chorea because of rare presentation.

Kaynakça

  • Brouns R, De Deyn PP. Neurological complications in renal failure: a review. Clin Neurol Neurosurg. 2004;107(1):1-16.
  • Laplane D, Baulac M, Widlocher D, Dubois B. Pure psychic akinesia with bilateral lesions of basal ganglia. J Neurol Neurosurg Psych,1984;47:377-85.
  • Wang HC, Cheng SJ. The syndrome of acute bilateral basal ganglia lesions in diabetic uremic patients. J. Neurol. 2003;250:948-55.
  • Lee EJ, Park JH, Ihn Y, Kim YJ, Lee SK, Park CS. Acute bilateral basal ganglia lesions in diabetic uraemia: diffusion-weighted MRI. Neuroradiology. 2007;49:1009-13.
  • Port JD, Beauchamp NJ, Jr. Reversible intracerebral pathologic entities mediated by vascular autoregulatory dysfunction. Radiographics .1998;18: 353Okada J, Yoshikawa K, Matsuo H, Kanno K, Oouchi M. Reversible MRI and CT findings in uremic encephalopathy. Neuroradiology. 1991;33:524-6.
  • Ertl-Wagner B, Jansen O, Schwap S, et al. Bilateral basal ganglion hemorrhage in diabetic ketoacidotic coma : Case report. Neuroradiology. 1999;41:670-3.
  • Brami-Zylberberg F, Meary E, Oppenheim C. Abnormalities of the basal ganglio and thalami in adults. J Radiol 2005;86:281-93.
  • Grippo J, Grippo T. Acute Encephalitis: Bilateral Lesions Of The Basal Ganglia. Rev Neurol. 2001;33:537-40.
  • Wang HC, Hsu JL, Shen YY: Acute bilateral basal ganglia lesions in patients with diabetic uremia: An FDG-PET study. Clin Nucl Med 2004;29:475-8.
  • Li JY, Yong TY, Sebben R, Khoo E, Disney AP: Bilateral basal ganglia lesions in patients with endstage diabetic nephropathy. Nephrology (Carlton). 2008;13:68-72.
  • Wang HC, Cheng SJ: The syndrome of acute bilateral basal ganglia lesions in diabetic uremic patients. J Neurol 2003;250:948-55.
  • Wang HC, Hsu JL, Shen YY. Acute Bilateral Basal Ganglia Lesions in Patients With Diabetic Uremia: An FDG-PET Study. Clin Nucl Med. 2004;29:475-8.
  • Altınkaya N, Yıldırım T, Yerdelen D. Acute Bilateral Basal Ganglia Lesions in Diabetic Uremic Patient: Imaging Findings. Turk Norol Derg. 2010;16:168-9.
  • Yazışma Adresi / Address for Correspondence: Dr. Fa ik İlik Elbistan Devlet Hastanesi Nöroloji Kliniği Elbistan/ KAHRAMANMARAŞ Email: faikilik@hotmail.com geliş tarihi/received :23.09.2013 kabul tarihi/accepted:21.10.2013

Diyabetik üremik hastada bazal ganglion tutulumu ve kore

Yıl 2014, Cilt: 39 Sayı: 2, 353 - 356, 22.07.2014
https://doi.org/10.17826/cutf.38301

Öz

Diyabetik üremik hastalarda akut bilateral bazal gangliyon oluşumu ile ilişkili sendromlar yaygın değildir. Üremik ensefalopati tipik olarak kortikal tutulum gösterir. Konfüzyon, nöbetler, tremor ve myoklonik nöbetler görülür. Bazal gangliyonlar anatomik olarak tutulursa kore dahil hareket bozuklukları görülür. Biz bu vakada koreye neden olan, bazal gangliyon tutulumlu üremik diyabetik hastayı nadir görülmesi nedeniyle sunduk.

Kaynakça

  • Brouns R, De Deyn PP. Neurological complications in renal failure: a review. Clin Neurol Neurosurg. 2004;107(1):1-16.
  • Laplane D, Baulac M, Widlocher D, Dubois B. Pure psychic akinesia with bilateral lesions of basal ganglia. J Neurol Neurosurg Psych,1984;47:377-85.
  • Wang HC, Cheng SJ. The syndrome of acute bilateral basal ganglia lesions in diabetic uremic patients. J. Neurol. 2003;250:948-55.
  • Lee EJ, Park JH, Ihn Y, Kim YJ, Lee SK, Park CS. Acute bilateral basal ganglia lesions in diabetic uraemia: diffusion-weighted MRI. Neuroradiology. 2007;49:1009-13.
  • Port JD, Beauchamp NJ, Jr. Reversible intracerebral pathologic entities mediated by vascular autoregulatory dysfunction. Radiographics .1998;18: 353Okada J, Yoshikawa K, Matsuo H, Kanno K, Oouchi M. Reversible MRI and CT findings in uremic encephalopathy. Neuroradiology. 1991;33:524-6.
  • Ertl-Wagner B, Jansen O, Schwap S, et al. Bilateral basal ganglion hemorrhage in diabetic ketoacidotic coma : Case report. Neuroradiology. 1999;41:670-3.
  • Brami-Zylberberg F, Meary E, Oppenheim C. Abnormalities of the basal ganglio and thalami in adults. J Radiol 2005;86:281-93.
  • Grippo J, Grippo T. Acute Encephalitis: Bilateral Lesions Of The Basal Ganglia. Rev Neurol. 2001;33:537-40.
  • Wang HC, Hsu JL, Shen YY: Acute bilateral basal ganglia lesions in patients with diabetic uremia: An FDG-PET study. Clin Nucl Med 2004;29:475-8.
  • Li JY, Yong TY, Sebben R, Khoo E, Disney AP: Bilateral basal ganglia lesions in patients with endstage diabetic nephropathy. Nephrology (Carlton). 2008;13:68-72.
  • Wang HC, Cheng SJ: The syndrome of acute bilateral basal ganglia lesions in diabetic uremic patients. J Neurol 2003;250:948-55.
  • Wang HC, Hsu JL, Shen YY. Acute Bilateral Basal Ganglia Lesions in Patients With Diabetic Uremia: An FDG-PET Study. Clin Nucl Med. 2004;29:475-8.
  • Altınkaya N, Yıldırım T, Yerdelen D. Acute Bilateral Basal Ganglia Lesions in Diabetic Uremic Patient: Imaging Findings. Turk Norol Derg. 2010;16:168-9.
  • Yazışma Adresi / Address for Correspondence: Dr. Fa ik İlik Elbistan Devlet Hastanesi Nöroloji Kliniği Elbistan/ KAHRAMANMARAŞ Email: faikilik@hotmail.com geliş tarihi/received :23.09.2013 kabul tarihi/accepted:21.10.2013
Toplam 14 adet kaynakça vardır.

Ayrıntılar

Birincil Dil Türkçe
Bölüm Araştırma
Yazarlar

Faik İlik Bu kişi benim

Hüseyin Büyükgöl Bu kişi benim

Ayşegül Öğmegül Bu kişi benim

Yayımlanma Tarihi 22 Temmuz 2014
Yayımlandığı Sayı Yıl 2014 Cilt: 39 Sayı: 2

Kaynak Göster

MLA İlik, Faik vd. “Diyabetik üremik Hastada Bazal Ganglion Tutulumu Ve Kore”. Cukurova Medical Journal, c. 39, sy. 2, 2014, ss. 353-6, doi:10.17826/cutf.38301.