ÇÜRÜK AKTİVİTE TESTLERİ (DERLEME)

Yıl 2020, Cilt: 30 Sayı: 2, 330 - 336, 15.04.2020

Ayberk İmren Mine Koruyucu Elif Bahar Tuna İnce Mehmet Tevfik Akıncı

https://doi.org/10.17567/ataunidfd.510000

Öz

Diş
çürüğü; mikroorganizma ve besinin diş yüzeyinde aynı zaman diliminde bulunması
ile bakterinin diş ile ilişkisinin bozulması sonucunda oluşmaktadır. Çürük
lezyonları klinik muayene ile incelenmekte ve tedavi edilmektedir. Ancak klinik
muayene çürük aktivitesini önceden tahmin edememektedir. Bu alanda yapılan
çalışmalar, günümüzde yoğun şekilde ilerlemektedir. Bu çalışmalardaki ana
hedef, kişilerin bireysel çürük aktivitesini ölçmek ve bunun sonucuna göre
koruyucu önlemler almaktır. Bu derlemede, günümüzde mevcut olan çürük aktivite
testleri ve cariogram programı hakkında bilgiler bulunmaktadır.

Anahtar Kelimeler: diş hekimliği, çürük
aktivite testleri, çürük risk faktörleri

CARIES ACTIVITY TESTS (REVIEW)

ABSTRACT

          Tooth
caries occurs when microorganism and nutrients are found in the same time zone
on the tooth surface, it is caused by the deterioration of the relation of the
bacteria with the tooth. Caries lesions are examined through clinical diagnoses
and treated. However, clinical diagnoses cannot predict caries in advance.
Studies in this area are continuing today. The goal of these studies is to
determine the predilection of people to caries and take the appropriate protective
precautions. In this review, there is information about caries activity tests
and cariogram program.

Key Words: dentistry, caries activity
test, risk factors of caries

Anahtar Kelimeler

diş hekimliği, çürük aktivite testleri

Kaynakça

• 1. Mandel ID. The role of saliva in maintaining oral homeostasis. The Journal of the American Dental Association 1989;119(2):298-304.
• 2. Moran J, Addy M, Newcombe R. A clinical trial to assess the efficacy of sanguinarine‐zinc mouthrinse (Veadent) compared with chlorhexidine mouthrinse (Corsodyl). Journal of clinical Periodontology 1988; 15(10):612-6.
• 3. World Health Organization. Review of methods of identification of high caries risk groups and individual. Federation Dentaire Internationale. Technical Report No. 31. Int Dent J 1988;38(3):1-3.
• 4. Taşveren SK, Akal N. Çürük Aktivite Testleri. Ondokuz Mayıs Üniversitesi Diş Hekimliği Fakültesi Dergisi 2015;7(1): 45-54.
• 5. Özgür B, Ünverdi GE, Çehreli Z. Diş Çürüğünün Tespitinde Geleneksel ve Güncel Yaklaşımlar. Turkiye Klinikleri Journal of Pediatric Dentistry-Special Topics 2018; 4(1): 1-9.
• 6. Gul P, Akgul N, Catakci AG, Atila A. Effects of of xylitol chewing gum, black tea and white cheese on saliva ph. The Journal of Dental Faculty of Atatürk University 2011; 21: 88-93.
• 7. Olszowski T, Adler G, Janiszewska‐Olszowska J, Safranow K, Kaczmarczyk M. MBL2, MASP2, AMELX, and ENAM gene polymorphisms and dental caries in Polish children. Oral diseases 2012;18(4): 389-395.
• 8. Featherstone JD, Domejean-Orliaguet S, Jenson L, Wolff M, Young DA. Caries risk assessment in practice for age 6 through adult. CDA 2007; 35(10): 710-3.
• 9. Edgar WM. Saliva: its secretion, composition and functions. British Dental Journal 1992;172(8): 305-12.
• 10. Sreebny LM. Saliva: Its role in health and disease. Working group 10 of the commission on oral health, research and epidemiology (CORE). Int Dent J. 1992;42:287-304.
• 11. Larmas M. Simple tests for caries susceptibility. International Dental Journal 1985;35(2): 109-117.
• 12. Kavanagh DA, Svehla G. Variation of salivary calcium, phosphate and buffering capacity in adolescents. Archives of Oral Biology 1998; 43(12):1023-1027.
• 13. Socransky SS, Manganiello SD. The oral microbiota of man from birth to senility. Journal of Periodontology 1971; 42(8):485-496
• 14. Ben-Aryeh H, Fisher M, Szargel R, Laufer D. Composition of whole unstimulated saliva of healthy children: changes with age. Archives of Oral Biology 1990;35(11):929-931.
• 15. Pandey P, Reddy NV, Rao VAP, Saxena A, Chaudhary CP. Estimation of salivary flow rate, pH, buffer capacity, calcium, total protein content and total antioxidant capacity in relation to dental caries severity, age and gender. Contemporary Clinical Dentistry 2015;6(1), S65.
• 16. Marsh PD. Microbial ecology of dental plaque and its significance in health and disease. Advances in Dental Research 1994; 8(2):263-271.
• 17. De Almeida PDV, Gregio AM., Machado MA, De Lima AA, Azevedo LR. Saliva composition and functions: a comprehensive review. J Contemp Dent Pract, 2008;9(3): 72-80.
• 18. Nikhila N, Arun A, Mythri H. Caries Activity Tests. Journal of Dental Sciences 2013; 1(3): 50-59.
• 19. De Soet JJ, Van Dalen PJ, Appelmelk BJ, De Graaff J. Identification of Streptococcus sobrinus with monoclonal antibodies. Journal of Clinical Microbiology 1987;25(12):2285-2288.
• 20. Van Houte J. Microbiological predictors of caries risk. Advances in Dental Research 1993;7(2): 87-96.
• 21. Emilson CG, Krasse BO. Support for and implications of the specific plaque hypothesis. European Journal of Oral Sciences 1985; 93(2):96-104.
• 22. Duru DÖ, Kırzıoğlu Z, Ayyıldız A. 3-6 yaş grubu çocuklarda çürük aktivite testleri ve çürük prevalansı arasındaki ilişki. Atatürk Üniversitesi Diş Hekimliği Fakültesi Dergisi 1999(2):3-6.
• 23. Russell JI, MacFarlane TW, Aitchison TC, Stephen KW, Burchell CK. Caries prevalence and microbiological and salivary caries activity tests in Scottish adolescents. Community Dentistry and Oral Epidemiology 1990;18(3):120-125.
• 24. Lemos JA, Burne RA. A model of efficiency: stress tolerance by Streptococcus mutans. Microbiology 2008; 154(11):3247-3255.
• 25. Kuramitsu HK, Wang BY. The whole is greater than the sum of its parts: dental plaque bacterial interactions can affect the virulence properties of cariogenic Streptococcus mutans. American Journal of Dentistry 2011; 24(3):153-154.
• 26. Camling E, Emilson CG. Results with the caries activity test" Cariostat" compared to prevalence of mutans streptococci and lactobacilli. Swedish Dental Journal 1989;13(4):125-130.
• 27. Schaeken MJM, Van der Hoeven JS, Franken HCM. Comparative recovery of Streptococcus mutans on five isolation media, including a new simple selective medium. Journal of Dental Research 1986; 65(6):906-908.
• 28. Fujiwara T, Sasada E, Mima N, Ooshima T. Caries prevalence and salivary mutans streptococci in 0–2‐year‐old children of Japan. Community Dentistry and Oral Epidemiology 1991;19(3): 151-154.
• 29. Suhonen J, Sener B, Bucher W, Lutz F. Release of preventive agents from pacifiers in vitro. An introduction to a novel preventive measure 1994;104(8): 946-951.
• 30. Alian AY, Mcnally M, Fure S, Birkhed D. Assessment of caries risk in elderly patients using the Cariogram model. Journal of the Canadian Dental Association 2006;72(5):459-63.
• 31. Bratthall D, Hänsel Petersson G Cariogram–a multifactorial risk assessment model for a multifactorial disease. Community Dentistry and Oral Epidemiology 2005;33(4):256-264.
• 32. Çelik EU, Gökay N, Ateş M. Efficiency of caries risk assessment in young adults using Cariogram. European Journal of Dentistry 2012;6(3): 270.
• 33. Bratthall D, Hänsel Petersson G, Stjernswärd JR. Cariogram manual. Cariogram, Internet Version 2.01. 2004.