Development of Cerebral Infarction After Stripping of Vena Saphena Magna: Case Report

Abstract

Cerebrovascular disease is the most frequent cause of neurological diseases. Embolic infarction is one of the most common causes of stroke. In the majority of patients with cerebral embolism, it is shown that emboli mostly originate from the heart via arterial circulation. Embolism rarely occurs as a paradoxical embolism through atrial / ventricular septal defect (ASD/VSD) or patent foramen ovale (PFO). In our clinical case, cerebral infarction is caused by ASD after vena saphena magna (VSM) stripping. Thirty- four- year- old female patient was admitted with motor and sensory loss at left lower extremity below the knee after stripping of VSM. Detailed neurological examination was done. She had left foot motor deficit, anesthesia of left lower extremity below the knee, impairment of motor strength (0/5 dorsiflexion and plantar flexion) of the left foot. After radiological investigation, diffusion weighted magnetic resonance imaging (MRI) revealed several, millimetric, nonspecific, nodular signal changes in some parts of bilateral periventricular, subcortical white matter of the brain that showing no diffusion limitation. In systematic examination, atrial septal defect was detected by echocardiography. Patient was treated with anticoagulant therapy. This case highlights detailed cardiological examination should be done in patients with cerebral infarction

Keywords

• Atrial septal defect, cerebral ischemia, lower extremity motor, sensory loss

Development of cerebral infarction after stripping of vena saphena magna

Öz

Kaynakça

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