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Urtica dioica L. attenuates bisphenol A-induced hepatotoxicity in rats by suppressing apoptosis and inflammation

Cilt: 51 Sayı: 1 2 Mart 2026
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Urtica dioica L. attenuates bisphenol A-induced hepatotoxicity in rats by suppressing apoptosis and inflammation

Öz

Purpose: The aim of this study was to investigate the protective effect of Urtica dioica L. (UD) against liver damage induced by bisphenol A (BPA) in rats. Materials and Methods: To this end, 48 healthy male Wistar albino rats were randomly divided into six groups: control (0.5 ml of normal saline); BPA (50 mg/kg); UD100 (100 mg/kg); UD200 (200 mg/kg); BPA+UD100; and BPA+UD200. All administrations were carried out for 56 days. The effects of the BPA and UD administrations on expression levels of Bcl-2-associated X protein (Bax), B-cell lymphoma 2 (Bcl-2) and tumor necrosis factor-α (TNF-α) in liver tissue samples were evaluated using Western blotting and immunohistochemical analyses. Results: BPA caused significant increases in the Bax/Bcl-2 expression ratio (+45.2-fold) and TNF-α levels (+12.2-fold) in liver tissue. In groups treated with UD at 100 and 200 mg/kg doses, apoptosis was suppressed by upregulating Bcl-2 expression and downregulating Bax expression. Furthermore, the co-administration of UD was observed to significantly reduce the histopathological damage caused by BPA in liver tissue. In the BPA+UD100 group, inflammation was suppressed by downregulating TNF-α expression (-18.8-fold); however, in the BPA+UD200 group, TNF-α expression levels were higher than in the control group. Conclusion: These findings suggest that UD treatment can mitigate the adverse effects of BPA on liver tissue and enhance tissue healing in rats.

Anahtar Kelimeler

Bisphenol A, Urtica dioica L., hepatotoxicity, apoptosis, inflammation

Destekleyen Kurum

Yazarlar herhangi bir mali destek almadıklarını beyan etmişlerdir.

Etik Beyan

Hatay Mustafa Kemal Üniversitesi Hayvan Deneyleri Yerel Etik Kurulu'ndan 24.06.2025 tarihli ve 2025/05-07 numaralı karar ile etik onay alınmıştır.

Kaynakça

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  2. Liu R, Liu B, Tian L, Jiang X, Li X, Cai D et al. Exposure to bisphenol a caused hepatoxicity and intestinal flora disorder in rats. Int J Mol Sci. 2022;23:8042.
  3. Shi R, Liu Z, Liu T. The antagonistic effect of bisphenol A and nonylphenol on liver and kidney injury in rats. Immunopharmacol Immunotoxicol. 2021;43:527-35.
  4. Fawzy HM, Negm EA. Effect of different doses of bisphenol a on albino rats' liver: histological and immunohistochemical study. Egypt J Histol. 2025;48:41-52.
  5. Abdulhameed ASAR, Lim V, Bahari H, Khoo BY, Abdullah MNH, Tan JJ et al. Adverse effects of bisphenol a on the liver and its underlying mechanisms: evidence from in vivo and in vitro studies. Biomed Res Int. 2022;2022:8227314.
  6. Mahdavinia M, Khorsandi L, Alboghobeish S, Samimi A, Dehghani MA, Zeidooni L. Liver histopathological alteration and dysfunction after bisphenol A administration in male rats and protective effects of naringin. Avicenna J Phytomed. 2021;11:394-406.
  7. Bindhumol V, Chitra KC, Mathur PP. Bisphenol A induces reactive oxygen species generation in the liver of male rats. Toxicology. 2003;188:117-24.
  8. Zaki A, Haidara MA, Abdallaa AM, Mohammed H, Sideeg AM, Eid RA. Role of dietary selenium in alleviating bisphenol A toxicity of liver albino rats: Histological, ultrastructural, and biomarker assessments. J Food Biochem. 2021;45:e13725.
  9. Wang K, Zhao Z, Ji W. Bisphenol A induces apoptosis, oxidative stress and inflammatory response in colon and liver of mice in a mitochondria-dependent manner. Biomed Pharmacother. 2019;117:109182.
  10. Tiegs G, Horst AK. TNF in the liver: targeting a central player in inflammation. Semin Immunopathol. 2022;44:445-59.

Kaynak Göster

APA
Pektanç Şengül, G., Kirman, G., & Şengül, S. A. (2026). Urtica dioica L. attenuates bisphenol A-induced hepatotoxicity in rats by suppressing apoptosis and inflammation. Cukurova Medical Journal, 51(1), 272-281. https://doi.org/10.17826/cumj.1784518
AMA
1.Pektanç Şengül G, Kirman G, Şengül SA. Urtica dioica L. attenuates bisphenol A-induced hepatotoxicity in rats by suppressing apoptosis and inflammation. Cukurova Med J. 2026;51(1):272-281. doi:10.17826/cumj.1784518
Chicago
Pektanç Şengül, Gülsüm, Günsel Kirman, ve Seydi Ahmet Şengül. 2026. “Urtica dioica L. attenuates bisphenol A-induced hepatotoxicity in rats by suppressing apoptosis and inflammation”. Cukurova Medical Journal 51 (1): 272-81. https://doi.org/10.17826/cumj.1784518.
EndNote
Pektanç Şengül G, Kirman G, Şengül SA (01 Mart 2026) Urtica dioica L. attenuates bisphenol A-induced hepatotoxicity in rats by suppressing apoptosis and inflammation. Cukurova Medical Journal 51 1 272–281.
IEEE
[1]G. Pektanç Şengül, G. Kirman, ve S. A. Şengül, “Urtica dioica L. attenuates bisphenol A-induced hepatotoxicity in rats by suppressing apoptosis and inflammation”, Cukurova Med J, c. 51, sy 1, ss. 272–281, Mar. 2026, doi: 10.17826/cumj.1784518.
ISNAD
Pektanç Şengül, Gülsüm - Kirman, Günsel - Şengül, Seydi Ahmet. “Urtica dioica L. attenuates bisphenol A-induced hepatotoxicity in rats by suppressing apoptosis and inflammation”. Cukurova Medical Journal 51/1 (01 Mart 2026): 272-281. https://doi.org/10.17826/cumj.1784518.
JAMA
1.Pektanç Şengül G, Kirman G, Şengül SA. Urtica dioica L. attenuates bisphenol A-induced hepatotoxicity in rats by suppressing apoptosis and inflammation. Cukurova Med J. 2026;51:272–281.
MLA
Pektanç Şengül, Gülsüm, vd. “Urtica dioica L. attenuates bisphenol A-induced hepatotoxicity in rats by suppressing apoptosis and inflammation”. Cukurova Medical Journal, c. 51, sy 1, Mart 2026, ss. 272-81, doi:10.17826/cumj.1784518.
Vancouver
1.Gülsüm Pektanç Şengül, Günsel Kirman, Seydi Ahmet Şengül. Urtica dioica L. attenuates bisphenol A-induced hepatotoxicity in rats by suppressing apoptosis and inflammation. Cukurova Med J. 01 Mart 2026;51(1):272-81. doi:10.17826/cumj.1784518