Araştırma Makalesi
BibTex RIS Kaynak Göster
Yıl 2024, Cilt: 51 Sayı: 2, 173 - 1181, 14.06.2024
https://doi.org/10.5798/dicletip.1501094

Öz

Kaynakça

  • 1.Nuki G, Simkin PA. A concise history of gout andhyperuricemia and their treatment. Arthritis ResTher. 2006; 8: 1.
  • 2.Dalbeth N, Gosling AL, Gaffo A, Abhishek A. Gout.Lancet. 2021; 397: 1843-55.
  • 3.Kuo C-F, Grainge MJ, Zhang W, Doherty M. Globalepidemiology of gout: prevalence, incidence and risk factors. Nat Rev Rheumatol. 2015; 11: 649–62.
  • 4.Dalbeth N, Merriman TR, Stamp LK. Gout. Lancet.2016; 388: 2039-52.
  • 5.Bellamy N, Downie WW, Buchanan WW.Observations on spontaneous improvement inpatients with podagra: implications for therapeutictrials of non-steroidal anti-inflammatory drugs. Br JClin Pharmacol. 1987; 24: 33–6.
  • 6.Grahame R, Scott JT. Clinical survey of 354patients with gout. Ann Rheum Dis. 1970; 29: 461–8
  • 7.Choi HK, Niu J, Neogi T, et al. Nocturnal risk of goutattacks. Arthritis Rheumatol. 2015; 67: 555–62.
  • 8.Singh JA, Herbey I, Bharat A, et al. Gout self-management in African American Veterans: aqualitative exploration of challenges and solutionsfrom patients’ perspectives. Arthritis Care Res. 2017; 69: 1724–32.
  • 9.Gaffo AL, Schumacher HR, Saag KG, et al.Developing a provisional definition of flare inpatients with established gout. Arthritis Rheum.2012; 64: 1508–17.
  • 10.Richette P, Doherty M, Pascual E, et al. 2018updated European League Against Rheumatismevidence-based recommendations for the diagnosisof gout. Annals of the rheumatic disease. 2020; 79:31-8.
  • 11.Maiuolo J, Oppedisano F, Gratteri S, Muscoli C,Mollace V. Regulation of uric acid metabolism andexcretion. Int J Cardiol. 2016; 213:8-14.
  • 12.World Health Organization. Obesity: preventingand managing the global epidemic. Report of a WHOconsultation. World Health Organ Tech Rep Ser.2000; 894-9.
  • 13.Taylor WJ, Fransen J, Jansen TL, et al. Study forupdated gout classification criteria: identification offeatures to classify gout. Arthritis Care Res. 2015;67: 1304–5.
  • 14.Reginato AJ, Schumacher HR Jr. Crystal-associated arthropathies. Clin Geriatr Med. 1988; 4:295–322.
  • 15.Schlesinger N, Watson DJ, Norquist JM. Serumurate during acute gout. J Rheumatol. 2009;36:1287–9.
  • 16.Brown BD, Hood Watson KL. Cellulitis. In:StatPearls. Treasure Island (FL): StatPearlsPublishing. 2023 Jan.
  • 17.Pérez-Garza DM, Chavez-Alvarez S, Ocampo-Candiani J, Gomez-Flores M. Erythema Nodosum: APractical Approach and Diagnostic Algorithm. Am JClin Dermatol. 2021; 22:367-78.
  • 18.Schlesinger N. Management of acute and chronicgouty arthritis: present state-of-the-art. Drug. 2004;64: 2399–416.
  • 19.Ahern MJ, Reid C, Gordon TP, et al. Doescolchicine work? The results of the first controlledstudy in acute gout. Aust NZJ Med.1987; 17: 301–4.
  • 20.Terkeltaub R, Furst D, Bennett K, et al. Highversus low dosing of oral colchicine for early acutegout flare. Arthritis Rheum. 2010; 62: 1060–8.

How well do we recognise gout disease?

Yıl 2024, Cilt: 51 Sayı: 2, 173 - 1181, 14.06.2024
https://doi.org/10.5798/dicletip.1501094

Öz

Objective: The clinical burden of gouty arthritis has historically been well recognized; however, gout is often misdiagnosed and mismanaged. In this study, we aimed to evaluate the diagnoses and treatments given to gout patients admitted to different specialties.
Methods: Patients who were diagnosed with gout attacks and treated by a rheumatologist were included, while patients with other non-gout rheumatic diseases (connective tissue diseases, rheumatoid arthritis, spondyloarthropathies, calcium pyrophosphate disease, etc.) were excluded. The branches the patients applied to during the attack, the treatments and diagnoses they received drugs, the number of attacks they had, demographic data, comorbidities, and laboratory data were recorded retrospectively.
Results: 424 gout patients were included. Patients were mostly male (70.7%). The mean age was 62.4± 12.4 years, and women were older than men (67.9±10 vs 62.4±12 years, p<0.001). Hypertension was the most common comorbidity, observed in 230 patients (54.2%). Among the patients who applied, 86 (20.2%) had previously been diagnosed, while 338 (79.7%) were diagnosed for the first time. The number of patients who had their first attack was 210 (49.5%), the number of patients who had their second attack was 88 (20.7%), and the number of patients who had ≥3 attacks was 126 (29.7%). The most commonly involved joint was the 1st metatarsophalangeal joint (MTF) and the second most commonly involved joint was the ankle joint. The rate of gout diagnosis was higher in patients presenting with podogra. The initial departments consulted during the incident were the emergency department first, followed by orthopedics and infectious diseases. Gout was the most common diagnosis, followed by trauma and injury, cellulitis, septic arthritis, and soft tissue infection. Nonsteroidal anti-inflammatory drugs(NSAIDs) were the most frequently prescribed drugs, followed by antibiotics and colchicine.
Conclusion: Gout is still not sufficiently recognized. Different diagnoses and treatments other than gout are made in applications to different branches. All physicians, regardless of their specialties, may be the first to see patients with gout attacks and therefore play a critical role in the diagnosis and treatment of these patients. With correct diagnosis and treatment, many visits to the doctor can be reduced.

Kaynakça

  • 1.Nuki G, Simkin PA. A concise history of gout andhyperuricemia and their treatment. Arthritis ResTher. 2006; 8: 1.
  • 2.Dalbeth N, Gosling AL, Gaffo A, Abhishek A. Gout.Lancet. 2021; 397: 1843-55.
  • 3.Kuo C-F, Grainge MJ, Zhang W, Doherty M. Globalepidemiology of gout: prevalence, incidence and risk factors. Nat Rev Rheumatol. 2015; 11: 649–62.
  • 4.Dalbeth N, Merriman TR, Stamp LK. Gout. Lancet.2016; 388: 2039-52.
  • 5.Bellamy N, Downie WW, Buchanan WW.Observations on spontaneous improvement inpatients with podagra: implications for therapeutictrials of non-steroidal anti-inflammatory drugs. Br JClin Pharmacol. 1987; 24: 33–6.
  • 6.Grahame R, Scott JT. Clinical survey of 354patients with gout. Ann Rheum Dis. 1970; 29: 461–8
  • 7.Choi HK, Niu J, Neogi T, et al. Nocturnal risk of goutattacks. Arthritis Rheumatol. 2015; 67: 555–62.
  • 8.Singh JA, Herbey I, Bharat A, et al. Gout self-management in African American Veterans: aqualitative exploration of challenges and solutionsfrom patients’ perspectives. Arthritis Care Res. 2017; 69: 1724–32.
  • 9.Gaffo AL, Schumacher HR, Saag KG, et al.Developing a provisional definition of flare inpatients with established gout. Arthritis Rheum.2012; 64: 1508–17.
  • 10.Richette P, Doherty M, Pascual E, et al. 2018updated European League Against Rheumatismevidence-based recommendations for the diagnosisof gout. Annals of the rheumatic disease. 2020; 79:31-8.
  • 11.Maiuolo J, Oppedisano F, Gratteri S, Muscoli C,Mollace V. Regulation of uric acid metabolism andexcretion. Int J Cardiol. 2016; 213:8-14.
  • 12.World Health Organization. Obesity: preventingand managing the global epidemic. Report of a WHOconsultation. World Health Organ Tech Rep Ser.2000; 894-9.
  • 13.Taylor WJ, Fransen J, Jansen TL, et al. Study forupdated gout classification criteria: identification offeatures to classify gout. Arthritis Care Res. 2015;67: 1304–5.
  • 14.Reginato AJ, Schumacher HR Jr. Crystal-associated arthropathies. Clin Geriatr Med. 1988; 4:295–322.
  • 15.Schlesinger N, Watson DJ, Norquist JM. Serumurate during acute gout. J Rheumatol. 2009;36:1287–9.
  • 16.Brown BD, Hood Watson KL. Cellulitis. In:StatPearls. Treasure Island (FL): StatPearlsPublishing. 2023 Jan.
  • 17.Pérez-Garza DM, Chavez-Alvarez S, Ocampo-Candiani J, Gomez-Flores M. Erythema Nodosum: APractical Approach and Diagnostic Algorithm. Am JClin Dermatol. 2021; 22:367-78.
  • 18.Schlesinger N. Management of acute and chronicgouty arthritis: present state-of-the-art. Drug. 2004;64: 2399–416.
  • 19.Ahern MJ, Reid C, Gordon TP, et al. Doescolchicine work? The results of the first controlledstudy in acute gout. Aust NZJ Med.1987; 17: 301–4.
  • 20.Terkeltaub R, Furst D, Bennett K, et al. Highversus low dosing of oral colchicine for early acutegout flare. Arthritis Rheum. 2010; 62: 1060–8.
Toplam 20 adet kaynakça vardır.

Ayrıntılar

Birincil Dil İngilizce
Konular Sağlık Kurumları Yönetimi, Tıp Eğitimi
Bölüm Original Articles
Yazarlar

Erdal Bodakçi

Yayımlanma Tarihi 14 Haziran 2024
Gönderilme Tarihi 15 Ocak 2024
Kabul Tarihi 24 Nisan 2024
Yayımlandığı Sayı Yıl 2024 Cilt: 51 Sayı: 2

Kaynak Göster

APA Bodakçi, E. (2024). How well do we recognise gout disease?. Dicle Tıp Dergisi, 51(2), 173-1181. https://doi.org/10.5798/dicletip.1501094
AMA Bodakçi E. How well do we recognise gout disease?. diclemedj. Haziran 2024;51(2):173-1181. doi:10.5798/dicletip.1501094
Chicago Bodakçi, Erdal. “How Well Do We Recognise Gout Disease?”. Dicle Tıp Dergisi 51, sy. 2 (Haziran 2024): 173-1181. https://doi.org/10.5798/dicletip.1501094.
EndNote Bodakçi E (01 Haziran 2024) How well do we recognise gout disease?. Dicle Tıp Dergisi 51 2 173–1181.
IEEE E. Bodakçi, “How well do we recognise gout disease?”, diclemedj, c. 51, sy. 2, ss. 173–1181, 2024, doi: 10.5798/dicletip.1501094.
ISNAD Bodakçi, Erdal. “How Well Do We Recognise Gout Disease?”. Dicle Tıp Dergisi 51/2 (Haziran 2024), 173-1181. https://doi.org/10.5798/dicletip.1501094.
JAMA Bodakçi E. How well do we recognise gout disease?. diclemedj. 2024;51:173–1181.
MLA Bodakçi, Erdal. “How Well Do We Recognise Gout Disease?”. Dicle Tıp Dergisi, c. 51, sy. 2, 2024, ss. 173-1181, doi:10.5798/dicletip.1501094.
Vancouver Bodakçi E. How well do we recognise gout disease?. diclemedj. 2024;51(2):173-1181.