COVID-19 and Diabetes - Prevalence and Prognosis: A Single-Center Experience
Öz
Anahtar Kelimeler
Kaynakça
- Reference1. Rothan HA, Byrareddy SN. The epidemiology and pathogenesis of coronavirus disease (COVID-19) outbreak. J Autoimmun. 2020;109:102433.
- Reference2. Poutanen SM. Etiologic agents of infectious diseases. In: Long SS, editor. Principles and practice of paediatric infectious diseases. 4th ed. 2012. p. 1547–712.
- Reference3. Zhang, H., Penninger, J. M., Li, Y., Zhong, N. & Slutsky, A. S. Angiotensin-converting enzyme 2 (ACE2) as a SARS-CoV-2 receptor: molecular mechanisms and potential therapeutic target. Intensive Care Med. 46, 586–590 (2020).
- Reference4. Huang, C. et al. Clinical features of patients infected with 2019 novel coronavirus in Wuhan, China. Lancet 395, 497–506 (2020).
- Reference5. Knapp S. Diabetes and infection:is there a link?- A mini review Gerontology, 2013;59:99-104.
- Reference6. Reza F, Nader B , Esfandiar H, et al. Serum levels of IL-32 in patients with type 2 diabetes mellitus and its relationship with TNF-α and IL-6.Cytokine 125 (2020) 154832.
- Reference7. Travis B.,Paul P., Jun Y, et al. Diabetes Exacerbates Infection via Hyperinflammation by Signaling through TLR4 and RAGE. mBio, 2017 8:e00818-17.
- Reference8. Allison L.T., Alan W., Sudhakar A., et al. Toll-Like Receptor 3 Signaling via TRIF Contributes to a Protective Innate Immune Response to Severe Acute Respiratory Syndrome Coronavirus Infection. mBio, 2015 , 3:e00638-15.
Ayrıntılar
Birincil Dil
İngilizce
Konular
Sağlık Kurumları Yönetimi
Bölüm
Araştırma Makalesi
Yayımlanma Tarihi
29 Ağustos 2021
Gönderilme Tarihi
27 Mart 2021
Kabul Tarihi
16 Haziran 2021
Yayımlandığı Sayı
Yıl 2021 Cilt: 5 Sayı: 2